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catarrhal and covered with a yellowish muco-purulent and greenish
coloured product; it is thickened and its sub-mucous tissue is infiltrated ;
its epithelium is desquamated, and it sometimes presents wounds with
loss of substance and covered with a pseudo membranous product.

On the laryngeal mucous membrane as also the tracheal and bron-
chial, we find the same alterations (hyperæmia, uniform or marbled
coloration, catarrhal condition, muco purulent products, etc.)

The lung is more or less hyperæmic, and its interlobular connective
tissue is the seat of a more or less abundant exudation. There is
pulmonary œdema and sometimes more or less extensive interlobular
emphysema. The bronchial ganglia are tumefied, hymeræmic, reddened
and softened.

The pleuræ have a reddish icteric tint either uniform or spotted;
they contain red coloured serosity.

The kidneys are sometimes tumefied, hyperæmic, softened and
friable. The vesical mucous membrane is injected and turgid. The
utero vaginal mucous membrane is congested, infiltrated, thickened
uniformly red as spotted and catarrhal. The mammary glands are flabby
and infiltrated.

The brain, spinal cord and nerves present more or less vivid hyperæ-
mia and an exudation into their connective tissue.

The lesions of the circulatory apparatus are little pronounced at the
commencement. The blood, at first but little changed, undergoes
important changes during the course of the malady. It becomes black
and loses its coagulability at the end.

The white corpuscles increase in number and " b onnets " are present
in it, the signification of which is at present not well understood. The
pericardium is congested, reddish coloured and spotted ; it contains sangui-
nolent serosity. The heart is yellow, pale coloured and softened ; the
endocardium offers ecchymosed patches, which also exist in the substance
of the heart ; the interior of the cardiac cavity and of the vessels is red
and icteric. In all organs the capillary network is relaxed, turgid, and
hyperæmic. The pathogeny of all the lesions is explained by the
alteration of the blood. The altered circulatory fluid acts on the
elements and on the vasculo motor nerves paralysing them; the vessels
then dilate and blood stasis occurs which results in the exudation,
infiltration, and dissolution of the coloured cells. At the surface of the
integument where exudation is active we observe a catarrhal state
accompanied by a ploriferation of the cellular elements and their rapid
degeneration.

Etiology contagion.—Rinderpest is a malady which always deve-
lops by contagion ; this is pretty well established at the present day;
it is absolutely false to say that it may develop spontaneously. In
Eastern countries like India it exists as an enzootic and perpetuates

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